The reasons for the cause rheumatic fever

Group A streptococcus rheumatic fever and wind on the etiology of heart disease between the epidemiological and clinical immunology in some indirect evidence has supported a number of clinical and epidemiological studies in group A streptococcus infection and rheumatic fever closely related to immunization study Also confirmed that before the onset of acute rheumatic fever there are early history of the streptococcus infection; forward-looking long-term follow-up of the anti-bacterial treatment and prevention of Streptococcus suis infection may be the primary prevention of rheumatic fever and relapse; means of infection is crucial Streptococcus Throat infection is the incidence of rheumatic fever must be conditions

Nevertheless Group A streptococcus cause rheumatic fever incidence has not yet understand the mechanism of Streptococcus rheumatic fever is not caused by direct infection because the incidence of rheumatic fever is not the streptococcus infection at the time but in the weeks after infection to the onset of rheumatic fever patients Culture and the blood in the heart tissue has never been found in the group A streptococcus after suffering from streptococcus pharyngitis only% ~% of patients in rheumatic fever

In recent years found that group A streptococcus cell wall containing protein for the MT-R and protein components of the M protein which can impede the most important role is swallowed up by type of bacteria, also known as the basis of “cross-antigen reaction” in the cell walls of Streptococcus Polysaccharide components of the species-specific antigen, also known as the “C substances” by the human Streptococcus suis infection may be some people not only to produce antibodies can be their own role in Streptococcus role in heart valve disease and valvular heart valve caused the mucopolysaccharide Composition with age and thus can explain variations in young people and adults of different heart valve disease incidence of immunology research suggested that the immune regulation of acute rheumatic fever there are shortcomings in their identity as B cells and helper T cells and inhibit the increased T-cell Led to relative decline in humoral and cellular immune enhancement is no chronic rheumatic heart disease rheumatoid activities but continued to exist prompted increased number of B-cell immune inflammatory process is still underway whether Streptococcus suis infection in rheumatic fever and also the response of the human body in this The level of response with the streptococcus antigen antibodies in the parallel relationship between allergy antibodies in a long time in the big opportunities; other hand, and the function of the nervous system related to the change

Related posts

• Youth Sports prevent anemia (0)
• Your triglycerides normal? (0)
• Your lips, and congenital heart disease Green (0)
• Young men renal protection measures (0)
• Women suffering from convulsions vulnerable to cardiovascular disease (0)
• Women slimming not deliberately Shoutui (0)
• Women should guard against excessive sex (0)
• Women often do not do housework uterine cancer risk (0)